A Stanford-led study of twins with and without food allergies has uncovered differences in the fecal bacteria of allergic and non-allergic individuals.
Stanford University researchers have developed a nanoparticle vaccine that has shown in mouse studies to effectively build coronavirus immunities.
Stanford scientists transformed tonsils into immunology labs in a dish, aiding research to develop vaccines for COVID-19, the flu and other diseases.
The Pfizer and Moderna COVID-19 vaccines are the first to use the RNA coding molecule to prompt our bodies to fight the virus. Here's how they work.
Understanding similarities between the Nipah virus and COVID-19 could provide clues for avoiding future novel virus outbreaks.
Stanford Medicine’s early development of testing for COVID-19 infection and antibodies helped guide government responses and stem local spread of the virus.
There's a voracious appetite for information on how SARS-CoV-2, the virus responsible for the COVID-19 pandemic, works. Here it is, in a single package.
How Stanford Medicine ramped up in the spring of 2020 to cope with a coming global pandemic and learned how to brace for the next wave of COVID-19 patients.
A Stanford research team is tasked with assessing the COVID-19 infection crisis inside California’s prisons and providing strategies to contain the virus.
Stanford researchers and colleagues have invented a genetic safety mechanism that can deactivate transplanted cells if they change in a problematic way.
Two recent Stanford-led studies show the value of tweaking vaccines to enlist the entire immune system — not just part of it — in preventing HIV infection.
A NOVA special featured Rhiju Das and the OpenVaccine project, in which gamers help scientists find an RNA molecule configuration for a COVID-19 vaccine.
Stanford scientists have taken important steps toward figuring out how to use immune therapy for a group of severe pediatric brain tumors.
The Stanford Clinical Virology Laboratory is ramping up capacity for its coronavirus diagnostic test, which can deliver results in 24 hours.
Scientists found a sneaky way to stop cold viruses from replicating in mammalian cells by disabling a protein not in the virus but in the cells they infect.
Mammalian cells use a label to distinguish self from non-self circular RNA molecules. Foreign molecules can trigger anti-cancer immune responses.