Scientists have modified immune cells, imbuing them with the ability to not only detect, but reveal, the presence of a tumor.
Cracking the crystal structure of a protein complex centered around a major immune signaling protein, interferon-gamma, may speed its medical use.
A new blood test measures the DNA fragments of lung transplant donors in the blood of recipients, in hopes of preventing organ rejection and saving lives.
Stanford scientists have moved a big step closer toward using engineered immune cells to treat many forms of pediatric cancer.
New research has confirmed that an antigen in some variants of the flu virus and vaccine can, in rare cases, trigger an autoimmune response leading to narcolepsy.
Sharon Chinthrajah weighs in on a new peanut allergy immunotherapy, speaking to its potential and its role in the future of food allergies therapy.
With age comes wisdom: mostly true. But a new study helps explain why one part of us - our immune system - gets decidedly dumber with age.
Much of what we know about the immune system comes from experiments conducted on mice. But lab mice are not little human beings. The two species are separated by both physiology and lifestyles. Stanford immunologist Mark Davis is calling on his colleagues to shift their research focus to people.
Hiding mRNA messages in CARTs — positively charged degradable vehicles —smuggles them across the cell membrane and can 'vaccinate' against cancer in mice.
The ratio between a certain types of immune cells is able to predict whether latent TB will shift into an active infection, new research has found.
Propionate molecules made by intestinal bacteria inhibits growth of Salmonella and may be a promising new treatment for gut infections.
A set of structurally similar proteins can activate a receptor for nicotine on immune cells, resulting in a dialing down of inflammation.
Scientists at Stanford find a biomarker for flu susceptibility, enabling predictions of if someone is going to fall ill to the virus after being exposed.
Older people are more susceptible to infection, cancer, and autoimmunity than younger people. This may be the result of our immune cells' receiving increasingly random marching orders as we age.
When associated with tumors, immune cells known as macrophages can be both good and bad: they can help cancer spread and curb its growth.
Rheumatoid arthritis and coronary artery disease share a common culprit: an important type of immune cell, called a macrophage, that has gone haywire. Stanford investigators have zeroed in on a molecular defect in macrophages' metabolic process that drives both disorders.
