A Stanford physician co-authored a list of likely biological factors underlying the reduced development of COVID-19 for children compared to adults.
Abdominal adhesions frequently occur after abdominal surgery. Stanford researchers prevented their formation in mice by blocking a molecular pathway.
Stanford-led research finds that the blood-brain barrier may be much more permeable -- albeit selectively so -- than previously thought.
Stanford researchers and colleagues have invented a genetic safety mechanism that can deactivate transplanted cells if they change in a problematic way.
Stanford researchers have found a good drug target for treating Diamond-Blackfan anemia, a genetic disease that impairs red blood cell formation.
Stanford scientists have devised a way to use positron emission tomography to watch the movement of a single cell injected into a lab mouse in real time.
How exactly does the antiviral drug remdesivir counter SARS-CoV-2 – the coronavirus strain responsible for COVID-19? And how well?
Stanford scientists have built a detailed picture of the biological clock of pregnancy, tracking thousands of metabolic markers throughout gestation.
A Stanford microbiologist describes the invigorating, yet sobering race to develop an effective vaccine against COVID-19.
Using microbubbles and ultrasound, researchers have created a cancer treatment that kills tumor cells and recruits immune cells to the tumor.
The experts on Stanford Medicine's molecular tumor board brainstorm new ways to attack individual patients' tumors at the genetic level.
Even if chloroquine and hydroxychloroquine don't end up being the best treatment for COVID-19, observing how they work in a dish can teach scientists a lot.
Research shows that misshapen proteins called prions can help yeast cells survive environmental threats, such as a lack of food and common antifungal drugs.
The discovery of a giant cavity in a key tuberculosis molecule could open the way for better understanding of the disease.
Co-infection with SARS-CoV-2 and other respiratory pathogens is more common than previously expected, according to a Stanford study.
A look at how viruses — including coronavirus — enter cells, use their molecular machinery to copy themselves and escape. And how to stop them.