Scientists have developed an algorithm that combines genome sequence data and electronic health information to predict risk for genetic disease.
John Farquhar, a beloved mentor, and pioneer in cardiovascular disease prevention at Stanford, died Aug. 22 at the age of 91.
Heart muscle cells from people with cardiomyopathies have shorter-than-normal telomeres -- the protective caps on chromosomes associated with aging.
For the past four years cardiologist Josh Knowles, MD, PhD, has been treating patients at Stanford who have a little-known but common genetic heart disease called …
Hypertrophic cardiomyopathy is caused by various genetic mutations that cause heart muscle to contract with too much force. New research suggests why.
This Stars of Stanford Medicine Q&A features Andrew Chang, clinical instructor of medicine, who is working to improve cardiovascular health globally.
Stanford researchers use gene editing and stem cell technologies to determine whether to worry — or not — about mysterious genetic test results.
In this final piece on aspirin for prevention of heart attack and stroke, Randall Stafford explains factors for doctors and patients to consider.
In this installment of "Aspirin for prevention," physician-researcher Randall Stafford provides tips to calculate the risk of heart disease or stroke, to inform decisions about taking aspirin preventatively.
A team of researchers has updated and improved the equations that guide prescribing decisions for physicians in the U.S. regarding cardiovascular risk.
Stanford heart doctors bank on digital health to improve heart care in the future by monitoring encouraging exercise, detecting and tracking conditions like atrial fibrillation, and more.
In this video, Stanford Medicine heart surgeon Joseph Woo discusses his award-winning research that examined the pros of cons of mechanical versus biological valve replacements.
Ben Thornton received a heart transplant when he was 3-years-old and later suffered a complication that left him struggling to walk. Now, he's thriving as a wheelchair basketball player.
Rheumatoid arthritis and coronary artery disease share a common culprit: an important type of immune cell, called a macrophage, that has gone haywire. Stanford investigators have zeroed in on a molecular defect in macrophages' metabolic process that drives both disorders.
A minimally-invasive procedure called TAVR "gave me back my life in an immediate and profound way," said Stanford high-risk heart patient Laura Hosking.
Stanford pediatric cardiologist Seth Hollander comments on Shaun White's success, and explains the condition called tetralogy of Fallot.