Scientists have modified immune cells, imbuing them with the ability to not only detect, but reveal, the presence of a tumor.
Cracking the crystal structure of a protein complex centered around a major immune signaling protein, interferon-gamma, may speed its medical use.
A team of researchers have found a new way to remove blood-producing stem cells, introducing the possibility of safer, and non-matched, transplants.
Sharon Chinthrajah weighs in on a new peanut allergy immunotherapy, speaking to its potential and its role in the future of food allergies therapy.
With age comes wisdom: mostly true. But a new study helps explain why one part of us - our immune system - gets decidedly dumber with age.
Geneticist Michael Snyder has tracked the expression of his genes for three years, focusing on changes in response to chronic or acute disease.
Much of what we know about the immune system comes from experiments conducted on mice. But lab mice are not little human beings. The two species are separated by both physiology and lifestyles. Stanford immunologist Mark Davis is calling on his colleagues to shift their research focus to people.
Hiding mRNA messages in CARTs — positively charged degradable vehicles —smuggles them across the cell membrane and can 'vaccinate' against cancer in mice.
Mapping the geography of the immune response in triple negative breast cancers predicts patient survival and sheds light onto new aspects of tumor biology.
A workaround avoids a common, dangerous side effect of gene therapy: an autoimmune reaction to the normal protein, which could improve gene therapy.
Scientists at Stanford find a biomarker for flu susceptibility, enabling predictions of if someone is going to fall ill to the virus after being exposed.
Exposure to 'third-hand smoke' — that is, the chemicals left behind on household surfaces after smoke has dissipated — increases the severity of asthma symptoms in mice. Stanford researchers are working to learn how this happens, and whether it might be possible to protect people with asthma from this exposure.
Older people are more susceptible to infection, cancer, and autoimmunity than younger people. This may be the result of our immune cells' receiving increasingly random marching orders as we age.
When associated with tumors, immune cells known as macrophages can be both good and bad: they can help cancer spread and curb its growth.
Rheumatoid arthritis and coronary artery disease share a common culprit: an important type of immune cell, called a macrophage, that has gone haywire. Stanford investigators have zeroed in on a molecular defect in macrophages' metabolic process that drives both disorders.
Many infectious diseases are marked by cyclical ups and downs. Stanford's David Schneider takes a creative approach to making sense of them.