On Wednesday, my colleague reported on a pair of recent Stanford studies that counteract the conventional belief that amyloid-forming proteins are universal enemies to the nervous system. As she explains, the research shows that these proteins are actually beneficial in animal models of multiple sclerosis.
Today on Science Friday, Lawrence Steinman, MD, professor of neurology and neurological sciences and of pediatrics at Stanford, discussed the work and why the studies “suggest the radical new idea that full-length, amyloid-forming proteins may in fact be produced by the body as a protective, rather than destructive, force.” A recording of the segment is now available online.
Previously: Amyloid, schmamyloid: Stanford MS expert finds dreaded proteins may not be all bad, Black hat in Alzheimer’s, white hat in multiple sclerosis? and Stanford neuroimmunologist discusses recent advances in MS research